Search results for "Memory impairment"

showing 10 items of 25 documents

Drosophila Full-Length Amyloid Precursor Protein is Required for Visual Working Memory and Prevents Age-Related Memory Impairment

2018

Summary The β-amyloid precursor protein (APP) plays a central role in the etiology of Alzheimer's disease (AD). However, its normal physiological functions are still unclear. APP is cleaved by various secretases whereby sequential processing by the β- and γ-secretases produces the β-amyloid peptide that is accumulating in plaques that typify AD. In addition, this produces secreted N-terminal sAPPβ fragments and the APP intracellular domain (AICD). Alternative cleavage by α-secretase results in slightly longer secreted sAPPα fragments and the identical AICD. Whereas the AICD has been connected with transcriptional regulation, sAPPα fragments have been suggested to have a neurotrophic and neu…

0301 basic medicineAgingFasciclin 2Nerve Tissue ProteinsGeneral Biochemistry Genetics and Molecular BiologyArticle03 medical and health sciences0302 clinical medicineAmyloid precursor proteinMemory impairmentAnimalsDrosophila ProteinsOlfactory memorybiologyWorking memoryfungiMembrane ProteinsLong-term potentiationCell biology030104 developmental biologyDrosophila melanogasterMemory Short-Termbiology.proteinVisual PerceptionAmyloid Precursor Protein SecretasesGeneral Agricultural and Biological SciencesAmyloid precursor protein secretase030217 neurology & neurosurgeryNeurotrophin
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Differential role of interleukin-1β in neuroinflammation-induced impairment of spatial and nonspatial memory in hyperammonemic rats.

2019

Activated microglia and increased brain IL-1β play a main role in cognitive impairment in much pathology. We studied the role of IL-1β in neuroinflammation-induced impairment of the following different types of learning and memory: novel object recognition (NOR), novel object location (NOL), spatial learning, reference memory (RM), and working memory (WM). All these processes are impaired in hyperammonemic rats. We assessed which of these types of learning and memory are restored by blocking the IL-1 receptor in vivo in hyperammonemic rats and the possible mechanisms involved. Blocking the IL-1 receptor reversed microglial activation in the hippocampus, perirhinal cortex, and prefrontal cor…

0301 basic medicineMaleInterleukin-1betaPostrhinal cortexHippocampusBiochemistryHippocampusReceptors N-Methyl-D-Aspartate03 medical and health sciences0302 clinical medicineReceptors GABAAmmoniaMemoryPerirhinal cortexGeneticsmedicineMemory impairmentAnimalsHyperammonemiaReceptors AMPARats WistarPrefrontal cortexMolecular BiologyNeuroinflammationCerebral CortexInflammationMicrogliabusiness.industryWorking memoryAnimal FeedRatsInterleukin 1 Receptor Antagonist ProteinProtein Subunits030104 developmental biologymedicine.anatomical_structureGene Expression RegulationReceptors GlutamateMicrogliabusinessNeuroscience030217 neurology & neurosurgeryBiotechnologyFASEB journal : official publication of the Federation of American Societies for Experimental Biology
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Indomethacin counteracts the effects of chronic social defeat stress on emotional but not recognition memory in mice

2017

We have previously observed the impairing effects of chronic social defeat stress (CSDS) on emotional memory in mice. Given the relation between stress and inflammatory processes, we sought to study the effectiveness of the anti-inflammatory indomethacin in reversing the detrimental effects of CSDS on emotional memory in mice. The effects of CSDS and indomethacin on recognition memory were also evaluated. Male CD1 mice were randomly divided into four groups: non-stressed + saline (NS+SAL); non-stressed + indomethacin (NS+IND); stressed + saline (S+SAL); and stressed + indomethacin (S+IND). Stressed animals were exposed to a daily 10 min agonistic confrontation (CSDS) for 20 days. All subjec…

0301 basic medicineMalemedicine.medical_treatmentEmotionsIndomethacinlcsh:MedicineSocial SciencesAnxietySocial defeatMice0302 clinical medicineCognitionLearning and MemoryMedicine and Health SciencesPsychologylcsh:ScienceSalineObject RecognitionMammalsCognitive ImpairmentMultidisciplinaryAnimal BehaviorBehavior AnimalCognitive NeurologyAnti-Inflammatory Agents Non-SteroidalNeurologyAnimal SocialityVertebratesAnxietymedicine.symptomResearch ArticleElevated plus mazemedicine.medical_specialtyCognitive NeurosciencePsychological StressRodents03 medical and health sciencesEmotionalityMemoryInternal medicineMental Health and PsychiatrymedicineAvoidance LearningMemory impairmentAnimalsInterpersonal RelationsRecognition memorySocial stressBehaviorbusiness.industrylcsh:RCognitive PsychologyOrganismsBiology and Life SciencesDisease Models Animal030104 developmental biologyEndocrinologyAmniotesChronic DiseaseCognitive Sciencelcsh:QPerceptionbusinessZoology030217 neurology & neurosurgeryStress PsychologicalNeurosciencePLoS ONE
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Confabulation: damage to a specific inferior medial prefrontal system

2008

Confabulation, the pathological production of false memories, occurs following a variety of aetiologies involving the frontal lobes, and is frequently held to be underpinned by combined memory and executive deficits. However, the critical frontal regions and specific cognitive deficits involved are unclear. Studies in amnesic patients have associated confabulation with damage to the orbital and ventromedial prefrontal cortices. However, neuroimaging studies have associated memory-control processes which are assumed to underlie confabulation with the right lateral prefrontal cortex. We used a confabulation battery to investigate the occurrence and localisation of confabulation in an unselect…

AdultMaleConfabulationDeceptionCognitive NeuroscienceConfabulation frontal lobe executive function memory orbitofrontal cortexVentromedial prefrontal cortexAmnesiaPrefrontal CortexExperimental and Cognitive PsychologyNeuropsychological TestsDelusionsFunctional Lateralityfrontal lobe.confabulation; frontal lobe; executive function; memory; orbitofrontal cortexmemoryNeuroimagingReference ValuesNeural PathwaysmedicineMemory impairmentHumansConfabulationEpisodic memoryAgedBrain MappingMiddle Agedfrontal lobeSelf ConceptNeuropsychology and Physiological Psychologymedicine.anatomical_structureFrontal lobeexecutive functionCase-Control StudiesOrbitofrontal cortexBrain Damage ChronicFemaleAmnesiamedicine.symptomPsychologyorbitofrontal cortexNeuroscience
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A Comparison of implicit memory tests in schizophrenic patients and normal controls

2007

The objective of the current study was to compare the performance of schizophrenic patients and normal controls on implicit memory tests. Two neuropsychological tasks were administered to 29 patients and normal participant samples. The implicit tests were: Word fragment completion and Word production from semantic categories. The priming score was the variable of interest. Priming effects are obtained in normal subjects and schizophrenia patients, regardless of the implicit test used. However, a dissociation in priming between normal and patient groups was observed, depending on the test used. For word fragment test, priming was identical between... (Ver más) the two groups. However, for wo…

AdultMaleLinguistics and LanguageDissociation (neuropsychology)Priming; Implicit memory; Schizophrenia; Memory impairmentUNESCO::PSICOLOGÍA::Psicología experimental::Procesos de la memoriaNeuropsychological TestsSeverity of Illness IndexVocabularyLanguage and Linguistics:PSICOLOGÍA::Psicología experimental::Procesos de la memoria [UNESCO]Surveys and QuestionnairesIndirect tests of memorymedicineHumansMemory impairmentMemory disorderNeuropsychological assessmentImplicit memoryGeneral PsychologyMemory Disordersmedicine.diagnostic_testCognitionMemory impairmentmedicine.diseaseSemanticsPrimingSchizophreniaFemaleImplicit memoryPsychologyPriming (psychology)Cognitive psychology
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A combined electrophysiological and morphological examination of episodic memory decline in amnestic mild cognitive impairment.

2013

Early stages of Alzheimer’s disease (AD) are characterized by neuropathological changes within the medial temporal lobe cortex (MTLC), which lead to characteristic impairments in episodic memory, i.e., amnestic mild cognitive impairment (aMCI). Here, we tested the neural correlates of this memory impairment using event-related potentials (ERPs) and voxel-based morphometry. Twenty-four participants were instructed to encode lists of words and were tested in a yes/no recognition memory task. The dual-process model of recognition memory dissociates between acontextual familiarity and recollection of contextual details. The early frontal ERP old/new-effect, which is thought to represent a neura…

AgingCognitive Neuroscienceevent-related potentialsrecognition memorybehavioral disciplines and activitieslcsh:RC321-571Event-related potentialMemory impairmentvoxel-based morphometryOriginal Research ArticleMedial Temporal Lobeslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryEpisodic memoryRecognition memoryfamiliarityRecallBrain morphometryevent-related potential (ERP)Voxel-based morphometrymedial temporal lobeVoxel Based MorphometryTemporal lobe/cortexPsychologyNeuroscienceCognitive psychologyNeuroscienceFrontiers in aging neuroscience
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Anosognosia and Metacognition in Alzheimer’s Disease

2015

While metacognition and anosognosia have long been studied as distinct concepts, more recently we have endeavored to construct a theoretical framework for exploring how metacognition can contribute to our understanding of anosognosia and vice versa. Following this approach, with a particular focus on Alzheimer’s disease (AD), this chapter first gives an overview of the key experimental findings and issues on metacognition in AD patients: in particular, overconfidence and absolute awareness; the sensitivity approach; the fractionation of metacognition in AD; the neural substrates of metacognition in the Alzheimer brain; and metacognitive control in these patients. Second, the chapter discuss…

AnosognosiaNeuropsychologymedicineMetacognitionMemory impairmentDiseasemedicine.diseasePsychologyCognitive psychology
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Analysis of Functional and Cognitive Impairment in Institutionalized Individuals with Movement Disorders

2018

Background: Many neurological disorders lead to institutionalization and can be accompanied in their advanced stages by functional impairment, and progressive loss of mobility, and cognitive alterations. Objective: We analyzed the relationship between functional impairment and cognitive performance and its related subdomains in individuals with Parkinson’s disease, Alzheimer’s disease accompanied by motor dysfunction, and with other neurological disorders characterized by both motor and cognitive problems. Methods: All participants lived in nursing homes (Valencia, Spain) and underwent cognitive evaluation with the Mini-Mental State Examination; functional assessment of independence in act…

Male0301 basic medicineMovement disordersActivities of daily livingParkinson's diseaseEndocrinology Diabetes and MetabolismOverweight03 medical and health sciences0302 clinical medicinemedicineHomes for the AgedHumansImmunology and AllergyMemory impairmentCognitive DysfunctionEffects of sleep deprivation on cognitive performanceAgedAged 80 and overCognitive evaluation theoryMovement Disordersbusiness.industryInstitutionalizationCognitionMental Status and Dementia Testsmedicine.diseaseNursing Homes030104 developmental biologySpainFemalemedicine.symptombusiness030217 neurology & neurosurgeryClinical psychologyEndocrine, Metabolic & Immune Disorders - Drug Targets
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Age differences in the role of the cannabinoid type 1 receptor on glutamatergic neurons in habituation and spatial memory acquisition

2015

Abstract Aims Aging is typically linked with a decline in memory performance and alterations in neural integrity. In pathological aging such as Alzheimer's disease, these effects are aggravated. Studies using cannabinoid CB1 receptor-deficient mice have shown a role of the endocannabinoid system in memory processing and neuroprotection. As the CB1 receptor is expressed in various neuronal populations, in this study, we aimed at investigating the consequences of CB1 receptor gene inactivation in cortical glutamatergic neurons in mice (Glu-CB1-KO) in regard to age-related alterations in spatial memory performance. Main methods Juvenile (5.5–7.5 weeks), adult (5.5–7 months), and old (11.5–14 m…

MaleAgingCannabinoid receptormedicine.medical_treatmentMorris water navigation taskBiologyGeneral Biochemistry Genetics and Molecular BiologyMiceGlutamatergicGlutamatesReceptor Cannabinoid CB1medicineAnimalsMemory impairmentGeneral Pharmacology Toxicology and PharmaceuticsHabituationHabituation PsychophysiologicMaze LearningSpatial MemoryMice KnockoutNeuronsThigmotaxisLearning DisabilitiesGeneral MedicineEndocannabinoid systemMice Inbred C57BLnervous systemlipids (amino acids peptides and proteins)CannabinoidNeurosciencePsychomotor Performancepsychological phenomena and processesLife Sciences
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Short-Term Long Chain Omega3 Diet Protects from Neuroinflammatory Processes and Memory Impairment in Aged Mice

2011

Regular consumption of food enriched in omega3 polyunsaturated fatty acids (oméga3 PUFAs) has been shown to reduce risk of cognitive decline in elderly, and possibly development of Alzheimer's disease. Docosahexaenoic acid (DHA) and eicosapentaenoic acid (EPA) are the most likely active components of oméga3-rich PUFAs diets in the brain. We therefore hypothesized that exposing mice to a DHA and EPA enriched diet may reduce neuroinflammation and protect against memory impairment in aged mice. For this purpose, mice were exposed to a control diet throughout life and were further submitted to a diet enriched in EPA and DHA during 2 additional months. Cytokine expression together with a thoroug…

MaleAnatomy and PhysiologyMouse[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionNeuroimmunologyNutritional Disorderslcsh:MedicineHippocampusHippocampusBiochemistryMiceLearning and Memory0302 clinical medicineImmune PhysiologyCognitive declinelcsh:Science2. Zero hungerchemistry.chemical_classification0303 health sciencesMultidisciplinaryFatty AcidsBrainfood and beveragesAnimal ModelsLipidsEicosapentaenoic acidmedicine.anatomical_structureEicosapentaenoic AcidNeurologyBiochemistryDocosahexaenoic acidAlimentation et NutritionCytokinesMedicineNeurogliaFemalelipids (amino acids peptides and proteins)Neurogenic InflammationResearch ArticlePolyunsaturated fatty acidmedicine.medical_specialtyDocosahexaenoic AcidsCognitive NeuroscienceImmunologyBiology03 medical and health sciencesModel OrganismsInternal medicineFatty Acids Omega-3medicineFood and NutritionAnimalsMemory impairmentWorking MemoryBiologyNeuroinflammationNutrition030304 developmental biologyInflammationMemory Disorderslcsh:RImmunityImmunologic SubspecialtiesDietMice Inbred C57BLNeuroanatomyEndocrinologychemistryDietary SupplementsClinical Immunologylcsh:Q[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryNeurosciencePLoS ONE
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